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Bioresorbable vascular scaffolds (BVS) allow the treatment of coronary lesions by offering vascular support and antiproliferative drug release without the need to implant a permanent metal layer in the coronary vessel. A problem occasionally observed during the follow-up is late fracture or late structural discontinuity(1,2). Although severe BVS dismantling has been associated with an increased risk of late BVS-thrombosis the implications of this phenomenon still remain unsettled(1,2). The use of BVS in patients with in-stent restenosis (ISR) has been demonstrated to be feasible, safe, and effective(3). However, implications of late BVS dismantling in patients treated for ISR currently ...
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